Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. Alternatively, our capability to rescue SERT operate in KI midbrain synaptoneurosomes via the inhibition of FAK indicates elevated FAK signaling downstream in the Pro32Pro33 mutant, as verified by amplified pFAK localization in 5-HT synapses. Our information https://crossu865xju6.blog2news.com/profile